Therapeutic effects of IGF-1 on stress urinary incontinence in rats with simulated childbirth trauma.
Identifieur interne : 000B92 ( Main/Exploration ); précédent : 000B91; suivant : 000B93Therapeutic effects of IGF-1 on stress urinary incontinence in rats with simulated childbirth trauma.
Auteurs : Yasuhiro Sumino [Japon] ; Satoru Yoshikawa [États-Unis] ; Hiromitsu Mimata [Japon] ; Naoki Yoshimura [États-Unis]Source :
- The Journal of urology [ 1527-3792 ] ; 2014.
Descripteurs français
- KwdFr :
- ARN messager (métabolisme), Accouchement (procédure) (effets indésirables), Animaux, Antigène nucléaire de prolifération cellulaire (métabolisme), Facteur de croissance IGF-I (usage thérapeutique), Femelle, Immunohistochimie, Incontinence urinaire d'effort (physiopathologie), Incontinence urinaire d'effort (traitement médicamenteux), Incontinence urinaire d'effort (étiologie), Modèles animaux de maladie humaine, Méthode TUNEL, Phosphorylation, Rat Sprague-Dawley, Rats, Transduction du signal (physiologie).
- MESH :
- effets indésirables : Accouchement (procédure).
- métabolisme : ARN messager, Antigène nucléaire de prolifération cellulaire.
- physiologie : Transduction du signal.
- physiopathologie : Incontinence urinaire d'effort.
- traitement médicamenteux : Incontinence urinaire d'effort.
- usage thérapeutique : Facteur de croissance IGF-I.
- étiologie : Incontinence urinaire d'effort.
- Animaux, Femelle, Immunohistochimie, Modèles animaux de maladie humaine, Méthode TUNEL, Phosphorylation, Rat Sprague-Dawley, Rats.
English descriptors
- KwdEn :
- Animals, Delivery, Obstetric (adverse effects), Disease Models, Animal, Female, Immunohistochemistry, In Situ Nick-End Labeling, Insulin-Like Growth Factor I (therapeutic use), Phosphorylation, Proliferating Cell Nuclear Antigen (metabolism), RNA, Messenger (metabolism), Rats, Rats, Sprague-Dawley, Signal Transduction (physiology), Urinary Incontinence, Stress (drug therapy), Urinary Incontinence, Stress (etiology), Urinary Incontinence, Stress (physiopathology).
- MESH :
- chemical , metabolism : Proliferating Cell Nuclear Antigen, RNA, Messenger.
- chemical , therapeutic use : Insulin-Like Growth Factor I.
- adverse effects : Delivery, Obstetric.
- drug therapy : Urinary Incontinence, Stress.
- etiology : Urinary Incontinence, Stress.
- physiology : Signal Transduction.
- physiopathology : Urinary Incontinence, Stress.
- Animals, Disease Models, Animal, Female, Immunohistochemistry, In Situ Nick-End Labeling, Phosphorylation, Rats, Rats, Sprague-Dawley.
Abstract
We examined the effect of IGF-1 in a rat model of stress urinary incontinence induced by simulated childbirth trauma.
DOI: 10.1016/j.juro.2013.08.109
PubMed: 24036237
Affiliations:
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Le document en format XML
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name="Pennsylvanie"><name sortKey="Yoshikawa, Satoru" sort="Yoshikawa, Satoru" uniqKey="Yoshikawa S" first="Satoru" last="Yoshikawa">Satoru Yoshikawa</name></region><name sortKey="Yoshimura, Naoki" sort="Yoshimura, Naoki" uniqKey="Yoshimura N" first="Naoki" last="Yoshimura">Naoki Yoshimura</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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